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675 Targeting Nrf2 has also become a research hotspot in TBI therapy due to its capacity to reduce oxidative stress and inhibit ferroptosis

Supporting this, data from our functional enrichment analysis of the transcriptome revealed notable upregulation of oxidative phosphorylation and the oxidative damage response in hepsin / mice treated with saline (data not shown)

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For example, ferroptotic macrophages have the ability to generate HMGB1, a DAMP that triggers the release of pro-inflammatory cytokines, including IL-1 and IL-18, and activates the inflammasome (61)

Long-term recurrent AR can lead to a decline in brain function and lack of concentration, reducing patients learning and working efficiency